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A sick child practically does not gain weight, his condition deteriorates rapidly, the skin turns pale, appetite decreases and weakness appears. A blood test shows leukopenia or leukocytosis with neutrophilia, hypochromic anemia with aniso- and poikilocytosis, sometimes monocytosis. Myelogram shows polymorphism of elavil marrow cells. The myelogram is characterized by polymorphism of bone marrow cells, sometimes the content of reticulohistiocytic elements is increased.

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FAQs

Abt-Letterer-Siwe disease is a disease of unknown etiology (cause), characterized by reticulohistiocytic proliferation (germination of pathological immune cells) in different organs of the body, which leads to their defeat. As a rule, children of the first or second year of life get sick. Symptoms PrCauses Diagnosis Treatment Complications and consequences Prevention. Symptoms of Abt-Letterer-Siwe disease.

The causes of the disease are unknown. It is assumed that the disease can be caused by bacteria and viruses that affected the pregnant or already born child, as well as ionizing, x-ray radiation.

amitriptyline examination of bones: determines the foci of destruction (destruction), osteoporosis (decrease in bone density). elavil examination of the lungs: reveals areas of swollen lung tissue, small blackouts in the form of nodules. It is also possible to consult an allergist-immunologist, hematologist, dermatologist, pulmonologist.

How long will I need to be in therapy?

Analysis of the anamnesis and complaints of the disease (according to the parents): how the disease began - with a sharp rise in body temperature; bleeding vesicles and nodules on the head and body; poor weight gain, loss of appetite; frequent infectious and inflammatory diseases.

General examination: the child is emaciated, pale, lethargic, the skin and mucous membranes are covered with bleeding nodules and scars;

enlarged lymph nodes (neck, head, in the groin), liver and spleen. Complete blood count: anemia is determined (a condition in which a reduced content of red blood cells or an insufficient amount of hemoglobin in them is observed in the bloodstream), lymphocytosis (an increase in immune cells in the blood), thrombocytopenia (a decrease in the number of elavil pills involved in the formation of blood clots in the process of blood clotting to stop bleeding), hyperleukocytosis (a significant increase in the number of leukocytes (white blood cells)).

Treatment of Abt-Letterer-Siwe disease.

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The appointment of glucocorticoids (drugs that inhibit the activity of immune cells). Contribute to the normalization of hematopoietic function, metabolism. The appointment of cytostatics (antineoplastic drugs) in order to stop the growth of histiocytes (cells that can absorb and digest small foreign elements) in the organs. Symptomatic treatment (aimed at reducing or eliminating symptoms).

Stomatitis (inflammation of the oral mucosa). Mastoiditis (inflammation of the mastoid process (part of the temporal bone located behind the ear)). Otitis. Gradually advancing cachexia (exhaustion of the body). Damage to the bones of the skull, limbs, pelvis, spine (decrease in their density, flattening of the vertebrae). Respiratory failure (a condition characterized by the inability of the respiratory system to provide the body with sufficient oxygen for life). The risk of death.

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The pediatrician will help in the treatment of the disease. Histological examination of the affected organs (lymph nodes, skin): accumulations of histiocytes (cells capable of absorbing and digesting small foreign elements) are detected in the punctate (a piece of tissue taken for analysis with a needle).

Abt-Letterer-Sive disease (true reticulohistiocytosis) was first described by Letterer in 1924. In 1933, Sive made a detailed report on the clinical picture, and in 1936 Abt summarized the results of his own research and literature data regarding acute reticulohistiocytosis in children. Therefore, the disease is called Abt-Letterer-Siwe disease. A number of synonyms for this disease are used in the literature: diffuse reticuloendotheliosis, aleukemic reticulosis, histiomonocytic acute malignant reticulosis, non-lipid reticulosis, acute reticuloendotheliosis, histiocytosis X, etc.

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